Urinary tract infections (UTIs) are the second most common bacterial infection with high recurrence rates in women1. With the rise of multi-drug resistant (MDR) bacteria, UTI treatment is becoming increasingly difficult, at great cost to healthcare systems2. Here we investigated whether protective adaptive immune responses occur in a mouse model of MDR UTI.
C57BL/6J wild-type or rag1-/-mice were transurethrally inoculated with an MDR UTI strain of Gram-negative uropathogenic Escherichia coli, depositing bacteria directly into the bladder3. Flow cytometry was used to characterise immune cell populations in bladder-draining lymph nodes of wild-type mice. Bladders and urine were also collected at various time-points to assess bacterial numbers.
In bladder-draining lymph nodes, germinal centre (GC) B cell responses had developed by 4 weeks post-infection, with immunoglobulin class switching towards IgG. GC B cell responses varied between individual infected wild-type mice. 30% of wild-type mice cleared bladder infection by 4 weeks, but we noted no clear correlation between the magnitude of GC B cell responses and bacterial load. At this timepoint, rag1-/-mice had significantly higher loads of bacteria in their bladders compared to wild-type mice, suggesting a role for adaptive immune responses in protecting against UTI. In conclusion, our data suggests for the first time in a mouse model, that Gram-negative bacterial UTI induces humoral immune responses in local lymph nodes draining the bladder, and that these responses could potentially help clear infection.