Introduction
Preterm Birth (PTB) is associated with 75% perinatal mortality and >50% long-term morbidity, such as neuro-developmental problems/ pulmonary dysfunction/ visual impairment. Infections and infection-driven inflammatory responses are considered as leading cause of PTB. Intrauterine infections can induce various stimulatory factors such as galectins/ proinflammatory cytokines/ chemokines/ prostaglandins which can trigger the early onset of labour and lead to PTB. Galectins are endogenous β-galactoside binding lectins, emerging as potential inflammatory mediator. Galectin-1 expresses at the feto-maternal interface and plays key role in maintenance of pregnancy by regulating the maternal immune response against placental alloantigens. Present study aimed to study in situ expression of galectin-1 in placenta of PTB patients infected with the pathogenic bacteria, Ureaplasma urealyticum (UU).
Materials and Methods
After obtaining hospital ethics committee permission, 35 patients (age = 19 - 31 years) undergoing premature delivery at Department of Obstetrics and Gynaecology, Safdarjung hospital, New Delhi, India were enrolled for collection of placental tissue (5cm x 5cm). Polymerase chain reaction assay was performed for UU detection by targeting MBA gene (364 bp) and for ruling out other sexually transmitted diseases (STD) pathogens. Real-time PCR was done for quantification of galectin-1 mRNA expression level by using commercial kit (Vivantis Technologies, Malaysia) as per manufacturer’s guidelines. Data was statistically analyzed by GraphPad Prism version 9.0.
Results
Placental UU was detected in 7 /35 (20%) of PTB patients. Galectin-1 expression was altered and found to be significantly (p < 0.05) upregulated in UU-positive PTB patients versus uninfected PTB patients (n = 28) with a fold change of 2.4.
Conclusions
Results suggest significant association of galectin-1 with PTB and its potential as a determinant for pathological pregnancies. Future studies targeting galectins in larger number of patients undergoing premature delivery are needed.